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阿司匹林对肠上皮细胞株Caco-2凋亡的影响(3)
http://www.100md.com 2011年12月1日 孙沂 张振玉 朱家沂 杨斌
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     [2]Thiefin G, Beaugerie L. Toxic effect of non-steroidal anti-inflammatory drugs on the small bowl, colon, and rectum[J]. Joint Bone Spine, 2005, 72(4):286-294.

    [3] Adebayo D, Bjarnason I. Is non-steroidal anti-inflammatory drug(NSAID) enteropathy clinically more important than NSAID gastropathy[J]? Postgrad Med J, 2006, 82(965):186-191.

    [4] Ono S, Kato M, Asaka M,et al.Diagnostic imaging of NSAID ulcers[J]. Nippon Rinsho, 2007, 65(10): 1792-1800.

    [5] Pozzoli C, Menozzi A, Grandi D,et al. Protective effects of proton pump inhibitors against indomethacin-induced lesions in the rat small intestine[J]. Naunyn Schmiedebergs Arch Pharmacol, 2007, 374(4): 283-291.

    [6] Fortun PJ, Hawkey CJ. Nonsteroidal antiinflammatory drugs and the small intestine[J]. Curr Opin Gastroenterol, 2007, 23(2): 134-141.

    [7] Alexey G, Anton M, Maciej S, et al. Kinetic modelling of NSAID action on COX-1: Focus on in vitro/in vivo aspects and drug combinations[J]. Eur J Pharm Sci, 2009, 36(1): 122-136.

    [8] Ajuebor MN, Singh A, Wallace JL. Cyclooxygenase-2-derived prostaglandin D(2) is an early anti-inflammatory signal in experimental colitis[J]. Am J Physiol Gastrointest Liver Physiol, 2000, 279(1):G238-244.

    [9] 安 敏, 张振玉. 阿司匹林对肠上皮细胞的损伤作用及机制[J]. 世界华人消化杂志, 2009, 17(13): 1355-1358.

    [10] Bjarnason I, Takeuchi K, Simpson R. NSAIDs: the Emperor's new dogma[J]? Gut, 2003, 52(9):1376-1378.

    [11] Rostom A, Dube C, Wells G, et al. Prevention of NSAID-induced gastroduodenal ulcers[J]. Cochrane Database Syst Rev, 2002, (4):CD002296.

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