右美托咪定减轻氧化应激改善H9C2细胞缺氧/复氧损伤(3)
[3]Hausenloy DJ，Yellon DM.Ischaemic conditioning and reperfusion injury[J].Nat Rev Cardiol，2016，13(4)：193-209.
[4]Morciano G，Giorgi C，Bonora M，et al.Molecular identity of the mitochondrial permeability transition pore and its role in ischemia-reperfusion injury[J].Journal of Molecular&Cellular Cardiology，2015，78(3)：142-153.
[5]Fischer TH，Herting J，Tirilomis T，et al.Ca2+/calmodulin-dependent protein kinase II and protein kinase A differentially regulate sarcoplasmic reticulum Ca+leak in human cardiac pathology [J].Circulation，2013，128(9)：970-981.
[6]Sovari AA.Cellular and Molecular Mechanisms of Arrhythmia by Oxidative Stress[J].Cardiology Research and Practice，2016，(3，article 59)：1-7.
[7]Yao YT，Liu DH，Li LH，et al.Comparison of cardio-preotective effects induced by different modalities of sevoflurane conditioning in isolated rat hearts[J].Perfusion，2016，31(2)：156-163.
[8]Guler L，Bozkirli F，Bedirli N，et al.Comparison of the effects of dexmedetomidine vs.Ketamin in cardiac ischemial reperfusion injury in rat-preliminary study[J].Adv Clin Exp Med，2014，23(5)：683-9.
[9]Terashima Y，Sato T，Yano T，et al.Roles of phospho-GSK-3β in myocardial protection afforded by activation of the mitochondrial K ATP channel[J].J Mol Cell Cardiol，2010，49(5)：762-770.
[10]Major J L，Salih M，Tuana B S.E2F6 protein levels modulate drug induced apoptosis in cardiomyocytes[J].Cellular Signalling，2017(40)：230-238.
收稿日期：2018-4-25；修回日期：2018-5-4
編辑/李桦, http://www.100md.com(李璟)
[4]Morciano G，Giorgi C，Bonora M，et al.Molecular identity of the mitochondrial permeability transition pore and its role in ischemia-reperfusion injury[J].Journal of Molecular&Cellular Cardiology，2015，78(3)：142-153.
[5]Fischer TH，Herting J，Tirilomis T，et al.Ca2+/calmodulin-dependent protein kinase II and protein kinase A differentially regulate sarcoplasmic reticulum Ca+leak in human cardiac pathology [J].Circulation，2013，128(9)：970-981.
[6]Sovari AA.Cellular and Molecular Mechanisms of Arrhythmia by Oxidative Stress[J].Cardiology Research and Practice，2016，(3，article 59)：1-7.
[7]Yao YT，Liu DH，Li LH，et al.Comparison of cardio-preotective effects induced by different modalities of sevoflurane conditioning in isolated rat hearts[J].Perfusion，2016，31(2)：156-163.
[8]Guler L，Bozkirli F，Bedirli N，et al.Comparison of the effects of dexmedetomidine vs.Ketamin in cardiac ischemial reperfusion injury in rat-preliminary study[J].Adv Clin Exp Med，2014，23(5)：683-9.
[9]Terashima Y，Sato T，Yano T，et al.Roles of phospho-GSK-3β in myocardial protection afforded by activation of the mitochondrial K ATP channel[J].J Mol Cell Cardiol，2010，49(5)：762-770.
[10]Major J L，Salih M，Tuana B S.E2F6 protein levels modulate drug induced apoptosis in cardiomyocytes[J].Cellular Signalling，2017(40)：230-238.
收稿日期：2018-4-25；修回日期：2018-5-4
編辑/李桦, http://www.100md.com(李璟)