microRNAs在心力衰竭分子通路中作用研究进展及中医药干预展望(5)
参见附件。
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[28] Duan Y，Zhou B，Su H，et al.miR-150 regulates high glucose-induced cardiomyocyte hypertrophy by targeting the transcriptional co-activator p300[J].Exp Cell Res，2013，319(3)：173-184.
[29] Shehadeh LA，Sharma S，Pessanha M，et al.MicroRNA-20a constrains p300-driven myocardial angiogenic transcription by direct targeting of p300[J].PLoS One，2013，8(11)：e79133.
[30] Li Q，Song XW，Zou J，et al.Attenuation of microRNA-1 derepresses the cytoskeleton regulatory protein twinfilin-1 to provoke cardiac hypertrophy[J].J Cell Sci，2010，123(Pt14)：2444-2452.
[31] Creemers EE，Pinto YM.Molecular mechanisms that control interstitial fibrosis in the pressure-overloaded heart[J].Cardiovasc Res，2011，89(2)：265-272.
[32] Roy S，Khanna S，Hussain SR，et al.MicroRNA expression in response to murine myocardial infarction：miR-21 regulates fibroblast metalloprotease-2 via phosphatase and tensin homologue[J].Cardiovasc Res，2009，82(1)：21-29.
[33] Cardin S，Guasch E，Luo X，et al.Role for MicroRNA-21 in atrial profibrillatory fibrotic remodeling associated with experimental postinfarction heart failure[J].Circ Arrhythm Electrophysiol，2012，5(5)：1027-1035.
[34] van Rooij E，Sutherland LB，Thatcher JE，et al.Dysregulation of microRNAs after myocardial infarction reveals a role of miR-29 in cardiac fibrosis[J].Proc Natl Acad Sci USA，2008，105(35)：13027-13032.
[35] Yang H.Genomics：A breakthrough point of modernization of traditional Chinese medicine[J].Medicine World，2000，1(8)：5-7.
[27] Sharma S，Liu J，Wei J，et al.Repression of miR-142 by p300 and MAPK is required for survival signaling via gp130 during adaptive hypertrophy[J].EMBO Mol Med，2012，4(7)：617-632.
[28] Duan Y，Zhou B，Su H，et al.miR-150 regulates high glucose-induced cardiomyocyte hypertrophy by targeting the transcriptional co-activator p300[J].Exp Cell Res，2013，319(3)：173-184.
[29] Shehadeh LA，Sharma S，Pessanha M，et al.MicroRNA-20a constrains p300-driven myocardial angiogenic transcription by direct targeting of p300[J].PLoS One，2013，8(11)：e79133.
[30] Li Q，Song XW，Zou J，et al.Attenuation of microRNA-1 derepresses the cytoskeleton regulatory protein twinfilin-1 to provoke cardiac hypertrophy[J].J Cell Sci，2010，123(Pt14)：2444-2452.
[31] Creemers EE，Pinto YM.Molecular mechanisms that control interstitial fibrosis in the pressure-overloaded heart[J].Cardiovasc Res，2011，89(2)：265-272.
[32] Roy S，Khanna S，Hussain SR，et al.MicroRNA expression in response to murine myocardial infarction：miR-21 regulates fibroblast metalloprotease-2 via phosphatase and tensin homologue[J].Cardiovasc Res，2009，82(1)：21-29.
[33] Cardin S，Guasch E，Luo X，et al.Role for MicroRNA-21 in atrial profibrillatory fibrotic remodeling associated with experimental postinfarction heart failure[J].Circ Arrhythm Electrophysiol，2012，5(5)：1027-1035.
[34] van Rooij E，Sutherland LB，Thatcher JE，et al.Dysregulation of microRNAs after myocardial infarction reveals a role of miR-29 in cardiac fibrosis[J].Proc Natl Acad Sci USA，2008，105(35)：13027-13032.
[35] Yang H.Genomics：A breakthrough point of modernization of traditional Chinese medicine[J].Medicine World，2000，1(8)：5-7.
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