三羟基异黄酮对肝纤维化大鼠血清和肝组织中TGF-β1表达的影响(3)
 |
| 第1页 |
参见附件(2417KB，3页)。
[3] Gressner AM, Weiskirchen R. Modern pathogenetic concepts of liver fibrosis suggest stellate cells and TGF-beta as major players and therapeutic targets. J Cell Mol Med,2006, 54(10): 76-99.
[4] 廖明, 李彦, 舒伟.三羟基异黄酮对大鼠肝星状细胞的增值及TGF-β1、MMP-2和TIMP-2表达的影响. 世界华人消化杂志,2010,18(3):229-233
[5] Giannelli G, Antonaci S. Immunological and molecular aspects of liver fibrosis in chronic hepatitis C virus infection. Histol Histopathol, 2005, 20(3): 939-944.
[6] Friedman SL. Mechanisms of disease: Mechanisms of hepatic fibrosis and therapeutic implications. Nat Clin Pract Gastroenterol Hepatol, 2004, 1(2): 98-105.
[7] Tsukada S, Parsons CJ, Rippe RA. Mechanisms of liver fibrosis. Clin Chim Acta,2006, 364(1): 33-60.
[8] Gressner AM, Weiskirchen R, Breitkopf K, et al. Roles of TGF-beta 1 in hepaticfibrosis. Front Biosci, 2002, 7(4): 793-807.
[9] Sanderson N, Factor V, Nagy J, et al. Hepatic expression of mature transforminggrowth factor beta 1 in transgenic mice results in multiple tissue lesions. Proc Natl Acad Sci USA, 1995, 92(7): 2572-2576.
[10] Kanzler S, Lohse AW, Keff A, et al.TGF-beta l in liver fibrosis: an inducible transgenic mouse model to study liver fibrogenesis. Am J Physiol Gastrointest Liver Physiol, 1999, 276(4): G1059-G1068.
[11] Schnur J, Olah J, Szepesi A, et al. Thorgeirsson SS. Thioacetamide-induced hepatic fibrosis in transforming growth factor beta-1 transgenic mice. Eur Gastroenterol Hepatol, 2004,16(2): 127-133.
[12] 高春芳,陆伦根.纤维化形成中的重要调控因子和相关因素,纤维化疾病的基础和临床.上海科学技术出版社,2004: 90-99.
您现在查看是摘要介绍页，详见PDF附件(2417KB，3页)。