aaMAPK信号通路与缺血再灌注心肌关系的研究进展(2)
参考文献
[1]JohnsDG， Ao Z， W illette RN， et al. Role of p38 MAP kinase in postcapillary venule leukocyte adhesion in- duced by ischemia /reperfusion injury. Pharmacol Res， 2005， 51： 463-471.
[2]Jaswal JS， GandhiM， Finegan BA， et al. Inhibition of p38 MAPK and AMPK restores adenosine-induced car- dioprotection in hearts stressed by antecedent ischemia by altering glucose utilization. Am J PhysiolHeartCirc Physio， 2007， 293：H1107-1114.
[3]BradyNR， Hamacher-Brady A， Gottlieb RA. Proapop- totic BCL-2 family members and mitochondrial dysfunc- tion during ischemia /reperfusion injury， a study emplo- ying cardiac HL-1 cells and GFP biosensors. Biochim BiophysActa， 2006，17(57)：667-678.
[5]WangM， Baker L， Tsai BM， et al. Sex differences in themyocardial inflammatory response to ischemia-reper- fusion injury. Am J Physiol EndocrinolMetab， 2005， 288：E321-326.
[5]RenJ，Zhang S，Kovacs A，et al.Role of p38a MAPKincardiac apoptosisandremodelingaftermyocardial infarction[J].JMol Cell Cardiol，2005，38(4)：617-623.
[6]SeeF，Thomas W，Way K，et al.p38 mitogen-activated protein kinaseinhibitionimprovescardiacfunctionandattenuatesleft ventricular remodelingfollowingmyocardial infarctionintherat[J].J AmColl Cardiol，2004，44(8)： 1679-1689. (王希 赵建力)
[1]JohnsDG， Ao Z， W illette RN， et al. Role of p38 MAP kinase in postcapillary venule leukocyte adhesion in- duced by ischemia /reperfusion injury. Pharmacol Res， 2005， 51： 463-471.
[2]Jaswal JS， GandhiM， Finegan BA， et al. Inhibition of p38 MAPK and AMPK restores adenosine-induced car- dioprotection in hearts stressed by antecedent ischemia by altering glucose utilization. Am J PhysiolHeartCirc Physio， 2007， 293：H1107-1114.
[3]BradyNR， Hamacher-Brady A， Gottlieb RA. Proapop- totic BCL-2 family members and mitochondrial dysfunc- tion during ischemia /reperfusion injury， a study emplo- ying cardiac HL-1 cells and GFP biosensors. Biochim BiophysActa， 2006，17(57)：667-678.
[5]WangM， Baker L， Tsai BM， et al. Sex differences in themyocardial inflammatory response to ischemia-reper- fusion injury. Am J Physiol EndocrinolMetab， 2005， 288：E321-326.
[5]RenJ，Zhang S，Kovacs A，et al.Role of p38a MAPKincardiac apoptosisandremodelingaftermyocardial infarction[J].JMol Cell Cardiol，2005，38(4)：617-623.
[6]SeeF，Thomas W，Way K，et al.p38 mitogen-activated protein kinaseinhibitionimprovescardiacfunctionandattenuatesleft ventricular remodelingfollowingmyocardial infarctionintherat[J].J AmColl Cardiol，2004，44(8)： 1679-1689. (王希 赵建力)