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《动脉硬化血栓血管生物学》.2004年.第8期
 * IL-8 Plasma Concentrations and the Risk of Future Coronary Artery Disease in Apparently Healthy Men and Women
 * C-Reactive Protein as a Screening Test for Cardiovascular Risk in a Multiethnic Population
 * Effects of Conventional or Lower Doses of Hormone Replacement Therapy in Postmenopausal Women
 * Cholesterol-Lowering Independent Regression and Stabilization of Atherosclerotic Lesions by Pravastatin and by Antimonocyte Chemoattractant
 * Interleukin-6, Fibrin D-Dimer, and Coagulation Factors VII and XIIa in Prediction of Coronary Heart Disease
 * Common Polymorphism in the MTP Promoter Attenuates the Dyslipidemic and Proatherogenic Effects of Excess Body Weight
 * Associations Among Plasma Lipoprotein Subfractions as Characterized by Analytical Capillary Isotachophoresis, Apolipoprotein E Phenotype, Al
 * Is There Really a Power Shortage in Clinical Trials Testing the "Homocysteine Hypothesis?"
 * Implications of Recent Clinical Trials for the National Cholesterol Education Program Adult Treatment Panel III Guidelines
 * Unique Morphology and Focal Adhesion Development of Valvular Endothelial Cells in Static and Fluid Flow Environments
 * Heat Shock Protein 90 Transfection Reduces Ischemia-Reperfusion–Induced Myocardial Dysfunction via Reciprocal Endothelial NO Synthase Serine
 * Smoking Cessation Rapidly Increases Circulating Progenitor Cells in Peripheral Blood in Chronic Smokers
 * Statins Upregulate PCSK9, the Gene Encoding the Proprotein Convertase Neural Apoptosis-Regulated Convertase-1 Implicated in Familial Hyperch
 * Apolipoprotein B100 Metabolism in Autosomal-Dominant Hypercholesterolemia Related to Mutations in PCSK9
 * Incidence of Obesity-Associated Cardiovascular Disease Is Related to Inflammation-Sensitive Plasma Proteins
 * Statins Upregulate PCSK9, the Gene Encoding the Proprotein Convertase Neural Apoptosis-Regulated Convertase-1 Implicated in Familial Hyperch
 * Subphysiologic Apolipoprotein E (ApoE) Plasma Levels Inhibit Neointimal Formation After Arterial Injury in ApoE-Deficient Mice
 * Endothelial Overexpression of Fas Ligand Decreases Atherosclerosis in Apolipoprotein E–Deficient Mice
 * Leukocyte-Derived Interleukin 10 Is Required for Protection Against Atherosclerosis in Low-Density Lipoprotein Receptor Knockout Mice
 * Matrix Metalloproteinase-9 Genotype Influences Large Artery Stiffness Through Effects on Aortic Gene and Protein Expression
 * Effects of Long-Term Daily Low-Dose Supplementation With Antioxidant Vitamins and Minerals on Structure and Function of Large Arteries
 * Circulating Oxidized Low-Density Lipoprotein and Its Association With Carotid Intima-Media Thickness in Asymptomatic Members of Familial Com
 * LRP1B Attenuates the Migration of Smooth Muscle Cells by Reducing Membrane Localization of Urokinase and PDGF Receptors
 * EGF-Like Domain of Tenascin-C Is Proapoptotic for Cultured Smooth Muscle Cells
 * Attenuation of Graft Arterial Disease by Manipulation of the LIGHT Pathway
 * Vasodilator-Stimulated Phosphoprotein Regulates Proliferation and Growth Inhibition by Nitric Oxide in Vascular Smooth Muscle Cells
 * Smad Expression in Human Atherosclerotic Lesions
 * Basic Fibroblast Growth Factor Antagonizes Transforming Growth Factor-?1–Induced Smooth Muscle Gene Expression Through Extracellular Signal–
 * Thrombomodulin-Protein C-EPCR System
 * Vascular Protection
 * Lysosomal Cysteine Proteases in Atherosclerosis
 * Proteolysis of the Pericellular Matrix
 * ATVB In Focus
 * RAGE Axis
 * The Mystery of PCSK9
 * How Does Mutant Proprotein Convertase Neural Apoptosis-Regulated Convertase 1 Induce Autosomal Dominant Hypercholesterolemia?
 * Aortic Valve
 * A Summary of Implications of Recent Clinical Trials for the National Cholesterol Education Program Adult Treatment Panel III Guidelines