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《动脉硬化血栓血管生物学》.2005年.第9期
 * Even Flow
 * Differential Antiatherogenic Effects of PPAR Versus PPAR Agonists
 * A Mouse Model of the Perimenopausal Transition
 * Atherosclerosis in Patients With Autoimmune Disorders
 * Rho-Kinase Is an Important Therapeutic Target in Cardiovascular Medicine
 * Heme Oxygenase and Atherosclerosis
 * Rapid Effects of Rosiglitazone Treatment on Endothelial Function and Inflammatory Biomarkers
 * Vascular Endothelium Has a Local Anti-Adenovirus Vector System and Glucocorticoid Optimizes Its Gene Transduction
 * Shear Stress Induces Endothelial Differentiation From a Murine Embryonic Mesenchymal Progenitor Cell Line
 * Peroxisome Proliferator-Activated Receptor Ligands Stimulate Endothelial Nitric Oxide Production Through Distinct Peroxisome Proliferator-A
 * Caveolin-1 Is Essential for Activation of Rac1 and NAD(P)H Oxidase After Angiotensin II Type 1 Receptor Stimulation in Vascular Smooth Muscl
 * Pentraxin 3 Inhibits Fibroblast Growth Factor 2–Dependent Activation of Smooth Muscle Cells In Vitro and Neointima Formation In Vivo
 * Signal-Crosstalk Between Rho/ROCK and c-Jun NH2-Terminal Kinase Mediates Migration of Vascular Smooth Muscle Cells Stimulated by Angiotensin
 * CD34+ Cells Home, Proliferate, and Participate in Capillary Formation, and in Combination With CD34– Cells Enhance Tube Formation in a 3-Dim
 * Identification and Characterization of Vascular Calcification–Associated Factor, a Novel Gene Upregulated During Vascular Calcification In V
 * Mast Cell Tryptase in Mast Cell Granules Enhances MCP-1 and Interleukin-8 Production in Human Endothelial Cells
 * Mechanisms of the Inhibitory Effect of Epigallocatechin-3-Gallate on Cultured Human Vascular Smooth Muscle Cell Invasion
 * Critical Role of Mst1 in Vascular Remodeling After Injury
 * Important Role of Apoptosis Signal-Regulating Kinase 1 in Ischemia-Induced Angiogenesis
 * Aldehyde Dehydrogenase 2 Plays a Role in the Bioactivation of Nitroglycerin in Humans
 * Heparin-Binding Epidermal Growth Factor–Like Growth Factor, Collateral Vessel Development, and Angiogenesis in Skeletal Muscle Ischemia
 * Overexpression of Human Apolipoprotein A-II in Transgenic Mice Does Not Impair Macrophage-Specific Reverse Cholesterol Transport In Vivo
 * Overexpression of Human Apolipoprotein A-II in Transgenic Mice Does Not Impair Macrophage-Specific Reverse Cholesterol Transport In Vivo
 * PPAR, but not PPAR, Activators Decrease Macrophage-Laden Atherosclerotic Lesions in a Nondiabetic Mouse Model of Mixed Dyslipidemia
 * Rosiglitazone Attenuates Atherosclerosis in a Model of Insulin Insufficiency Independent of Its Metabolic Effects
 * Atherosclerotic Lesion Development in a Novel Ovary-Intact Mouse Model of Perimenopause
 * Genetic Deletion or Antibody Blockade of 1?1 Integrin Induces a Stable Plaque Phenotype in ApoE–/– Mice
 * Association Between Prostaglandin E Receptor Subtype EP4 Overexpression and Unstable Phenotype in Atherosclerotic Plaques in Human
 * An Oral ApoJ Peptide Renders HDL Antiinflammatory in Mice and Monkeys and Dramatically Reduces Atherosclerosis in Apolipoprotein E–Null Mice
 * Cytoplasmic Expression and Extracellular Deposition of an Antiangiogenic Factor, Pigment Epithelium-Derived Factor, in Human Atherosclerotic
 * Tendon Xanthomas in Familial Hypercholesterolemia Are Associated With Cardiovascular Risk Independently of the Low-Density Lipoprotein Recep
 * Effects of Diet and Simvastatin on Fatty Acid Composition in Hypercholesterolemic Men
 * Cytosolic Lipid Droplets Increase in Size by Microtubule-Dependent Complex Formation
 * Relationship of Monocyte Count and Peripheral Arterial Disease
 * Hepatic and Cardiovascular Consequences of Familial Hypobetalipoproteinemia
 * The Molecular Basis of Lecithin:Cholesterol Acyltransferase Deficiency Syndromes
 * Familial Combined Hyperlipidemia in Mexicans
 * Recurrence Rate After a First Venous Thrombosis in Patients With Familial Thrombophilia
 * Protective Mechanisms of Inosine in Platelet Activation and Cerebral Ischemic Damage
 * Endogenous NO Blockade Enhances Tissue Factor Expression via Increased Ca2+ Influx Through MCP-1 in Endothelial Cells by Monocyte Adhesion
 * Consequences of Cholesteryl Ester Transfer Protein Inhibition in Patients With Familial Hypoalphalipoproteinemia
 * Toll-Like Receptors, Endocrine Stress Response, and Arteriosclerosis
 * Lipopolysaccharide (LPS) Contamination Plays the Real Role in C-Reactive Protein–Induced IL-6 Secretion From Human Endothelial Cells In Vitr
 * Association of C-Reactive Protein With Blood Pressure