《高血压学杂志》.2006年.第4期
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- Rapid Nongenomic Effects of Aldosterone on the Renal Vasculature in Humans
- Heart Failure and Left Ventricular Remodeling After Reperfused Acute Myocardial Infarction in Patients With Hypertension
- The Positive Inotropic Effect of Angiotensin II
- Deletion of Angiotensin-Converting Enzyme 2 Accelerates Pressure Overload-Induced Cardiac Dysfunction by Increasing Local Angiotensin II
- Targeted Deletion of Matrix Metalloproteinase 2 Ameliorates Myocardial Remodeling in Mice With Chronic Pressure Overload
- Postischemic Brain Injury Is Exacerbated in Mice Lacking the Kinin B2 Receptor
- Gene Transfer of Hepatocyte Growth Factor Gene Improves Learning and Memory in the Chronic Stage of Cerebral Infarction
- Coexpression of Voltage-Dependent Calcium Channels Cav1.2, 2.1a, and 2.1b in Vascular Myocytes
- Reactivity of Ambulatory Blood Pressure to Physical Activity Varies With Time of Day
- Value of Noninvasive Hemodynamics to Achieve Blood Pressure Control in Hypertensive Subjects
- Cytochrome P450 Epoxygenase Gene Function in Hypoxic Pulmonary Vasoconstriction and Pulmonary Vascular Remodeling
- Long-Term Intake of North American Ginseng Has No Effect on 24-Hour Blood Pressure and Renal Function
- Mineralocorticoid Receptor Antagonism Attenuates Cardiac Hypertrophy and Failure in Low-Aldosterone Hypertensive Rats
- Brachial Blood Pressure But Not Carotid Arterial Waveforms Predict Cardiovascular Events in Elderly Female Hypertensives
- Cardioprotective Mechanisms of Eplerenone on Cardiac Performance and Remodeling in Failing Rat Hearts
- Cardioprotective Effects of Eplerenone in the Rat Heart
- Genome-Wide Linkage Study of Retinal Vessel Diameters in the Beaver Dam Eye Study
- NADPH Oxidase in the Renal Medulla Causes Oxidative Stress and Contributes to Salt-Sensitive Hypertension in Dahl S Rats
- Impaired Capacity for Stimulated Fibrinolysis in Primary Hypertension Is Restored by Antihypertensive Therapy
- Hypertension and Sympathoexcitation Driven by Increased NaCl Levels
- Angiotensin II Type 1 Receptor Blockers Reduce Urinary Oxidative Stress Markers in Hypertensive Diabetic Nephropathy