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《糖尿病学杂志》.2005年.第1期
 * Treatment With Granulocyte ColonyeCStimulating Factor Prevents Diabetes in NOD Mice by Recruiting Plasmacytoid Dendritic Cells and Functiona
 * Perspectives in Diabetes
 * Cell Function Following Human Islet Transplantation for Type 1 Diabetes
 * Defective Suppressor Function in CD4+CD25+ T-Cells From Patients With Type 1 Diabetes
 * Hyperglycemia Induces Monocytic Release of Interleukin-6 via Induction of Protein Kinase C- and -
 * Nuclear Factor-B Activity in -Cells Is Required for Glucose-Stimulated Insulin Secretion
 * Uncoupling of Nutrient Metabolism From Insulin Secretion by Overexpression of Cytosolic Phospholipase A2
 * Autoimmune Diabetes and Resistance to Xenograft Transplantation Tolerance in NOD Mice
 * Glucagon-like Peptide 1 Can Directly Protect the Heart Against Ischemia/Reperfusion Injury
 * Dynamic Changes in -Cell Mass and Pancreatic Insulin During the Evolution of Nutrition-Dependent Diabetes in Psammomys obesus
 * Genetic Background Determines the Size and Structure of the Endocrine Pancreas
 * Physical Activity in the Prevention of Type 2 Diabetes
 * Delayed Transcapillary Delivery of Insulin to Muscle Interstitial Fluid After Oral Glucose Load in Obese Subjects
 * Deficiency of Subsarcolemmal Mitochondria in Obesity and Type 2 Diabetes
 * Impairment of Host Resistance to Listeria monocytogenes Infection in Liver of db/db and ob/ob Mice
 * Insulin Deficiency Downregulated Heat Shock Protein 60 and IGF-1 Receptor Signaling in Diabetic Myocardium
 * Predictors of and Longitudinal Changes in Insulin Sensitivity and Secretion Preceding Onset of Type 2 Diabetes
 * Postnatal Diet-Induced Obesity in Rats Upregulates Systemic and Adipose Tissue Glucocorticoid Metabolism During Development and in Adulthood
 * Evidence for Accelerated Rates of Glutathione Utilization and Glutathione Depletion in Adolescents With Poorly Controlled Type 1 Diabetes
 * Predictors of and Longitudinal Changes in Insulin Sensitivity and Secretion Preceding Onset of Type 2 Diabetes
 * a-Series Gangliosides Mediate the Effects of Advanced Glycation End Products on Pericyte and Mesangial Cell Proliferation
 * Nitrergic Neurodegeneration in Cerebral Arteries of Streptozotocin-Induced Diabetic Rats
 * The Effect of Vitamin E on Endothelial Function of Micro- and Macrocirculation and Left Ventricular Function in Type 1 and Type 2 Diabetic P
 * Decreased Expression of Pigment Epithelium-Derived Factor Is Involved in the Pathogenesis of Diabetic Nephropathy
 * Aldose Reductase Inhibition Counteracts Oxidative-Nitrosative Stress and Poly(ADP-Ribose) Polymerase Activation in Tissue Sites for Diabetes
 * Altered Transcapillary Escape of Albumin and Microalbuminuria Reflects Two Different Pathogenetic Mechanisms
 * Differential Effects of Glucose and Lactate on Glucosensing Neurons in the Ventromedial Hypothalamic Nucleus
 * Linkage of Plasma Adiponectin Levels to 3q27 Explained by Association With Variation in the APM1 Gene
 * Genetic Linkage and Association of the Growth Hormone Secretagogue Receptor (Ghrelin Receptor) Gene in Human Obesity
 * Development of a Long-Acting Insulin Analog Using Albumin Fusion Technology
 * A Genome Scan for Fasting Insulin and Fasting Glucose Identifies a Quantitative Trait Locus on Chromosome 17p
 * Common Polymorphisms in the Adiponectin Gene ACDC Are Not Associated With Diabetes in Pima Indians
 * Heritability of Insulin Secretion, Peripheral and Hepatic Insulin Action, and Intracellular Glucose Partitioning in Young and Old Danish Twi
 * Increased Phosphorylation of Akt Substrate of 160 kDa (AS160) in Rat Skeletal Muscle in Response to Insulin or Contractile Activity
 * Increased Glucocorticoid Receptor and 11-Hydroxysteroid Dehydrogenase Type 1 Expression in Hepatocytes May Contribute to the Phenotype of Ty
 * HIV-1 Accessory Protein Vpr Inhibits the Effect of Insulin on the Foxo Subfamily of Forkhead Transcription Factors by Interfering With Their
 * Islet-infiltrating B-Cells in Nonobese Diabetic Mice Predominantly Target Nervous System Elements
 * Enhanced Hypothalamic AMP-Activated Protein Kinase Activity Contributes to Hyperphagia in Diabetic Rats
 * Growth Hormone Overexpression in the Central Nervous System Results in Hyperphagia-Induced Obesity Associated With Insulin Resistance and Dy