现在位置：百拇书刊 > 国外期刊 > 糖尿病学杂志 > 2005年.第9期 《糖尿病学杂志》.2005年.第9期  * Differentiation of Human Liver-Derived, Insulin-Producing Cells Toward the -Cell Phenotype  * New Clinical Syndromes, New Scientific Insights, and New Therapy  * Combination Therapy With Epidermal Growth Factor and Gastrin Increases -Cell Mass and Reverses Hyperglycemia in Diabetic NOD Mice  * Conditional Expression Demonstrates the Role of the Homeodomain Transcription Factor Pdx1 in Maintenance and Regeneration of -Cells in the A  * Essential Role for Membrane Lipid Rafts in Interleukin-1eCInduced Nitric Oxide Release From Insulin-Secreting Cells  * Glucagon-Like Peptide 1 Regulates Sequential and Compound Exocytosis in Pancreatic Islet -Cells  * Matrix Metalloproteinases Contribute to Insulin Insufficiency in Zucker Diabetic Fatty Rats  * Exposure to Chronic High Glucose Induces -Cell Apoptosis Through Decreased Interaction of Glucokinase With Mitochondria  * Marked Increase in White Adipose Tissue Blood Perfusion in the Type 2 Diabetic GK Rat  * Chronic Hyperglycemia, Independent of Plasma Lipid Levels, Is Sufficient for the Loss of -Cell Differentiation and Secretory Function in the  * Transgenic Mouse Overexpressing Syntaxin-1A as a Diabetes Model  * Adiponectin Is Lower Among African Americans and Is Independently Related to Insulin Sensitivity in Children and Adolescents  * Experimental Diabetes Attenuates Cerebral CorticaleCEvoked Forelimb Motor Responses  * Cardiac-Specific Overexpression of Peroxisome ProliferatoreCActivated Receptor- Causes Insulin Resistance in Heart and Liver  * Type 2 Diabetes Causes Remodeling of Cerebrovasculature via Differential Regulation of Matrix Metalloproteinases and Collagen Synthesis  * Characterization of Susceptibility of Inbred Mouse Strains to Diabetic Nephropathy  * Activation of Peripheral Blood CD14+ Monocytes Occurs in Diabetes  * Variation in the eNOS Gene Modifies the Association Between Total Energy Expenditure and Glucose Intolerance  * Genome-Wide Linkage Analyses of Type 2 Diabetes in Mexican Americans  * ATP and Sulfonylurea Sensitivity of Mutant ATP-Sensitive K+ Channels in Neonatal Diabetes  * Acute Bidirectional Manipulation of Muscle Glucose Uptake by In Vivo Electrotransfer of Constructs Targeting Glucose Transporter Genes  * Haptoglobin Polymorphism Predicts 30-Day Mortality and Heart Failure in Patients With Diabetes and Acute Myocardial Infarction  * Co-occurrence of Two Partially Inactivating Polymorphisms of MC3R Is Associated With Pediatric-Onset Obesity  * INS VNTR Class Genotype and Indexes of Body Size and Obesity  * The Functional Q84R Polymorphism of Mammalian Tribbles Homolog TRB3 Is Associated With Insulin Resistance and Related Cardiovascular Risk in  * Spillover of Dietary Fatty Acids and Use of Serum Nonesterified Fatty Acids for the Synthesis of VLDL-Triacylglycerol Under Two Different Fe  * Colocalization of Mouse Autoimmune Diabetes Loci Idd21.1 and Idd21.2 With IDDM6 (Human) and Iddm3 (Rat)  * Lack of Association of PAX4 Gene With Type 1 Diabetes in the Finnish and Hungarian Populations  * Expression of Mfn2, the Charcot-Marie-Tooth Neuropathy Type 2A Gene, in Human Skeletal Muscle  * Overactivation of S6 Kinase 1 as a Cause of Human Insulin Resistance During Increased Amino Acid Availability  * Changes of Adiponectin Oligomer Composition by Moderate Weight Reduction  * Increased Dietary Substrate Delivery Alters Hepatic Fatty Acid Recycling in Healthy Men  * Novel Leptin Receptor Mutation in NOD/LtJ Mice Suppresses Type 1 Diabetes Progression  * Increased Fat Mass Compensates for Insulin Resistance in Abdominal Obesity and Type 2 Diabetes  * The X-Linked Inhibitor of Apoptosis Protein Enhances Survival of Murine Islet Allografts  * Rosiglitazone Improves Myocardial Glucose Uptake in Patients With Type 2 Diabetes and Coronary Artery Disease  * Interferon- as a Mediator of Polyinosinic:Polycytidylic AcideCInduced Type 1 Diabetes  * XIAP Overexpression in Human Islets Prevents Early Posttransplant Apoptosis and Reduces the Islet Mass Needed to Treat Diabetes  * Very Slow Turnover of -Cells in Aged Adult Mice  * LEW.1WR1 Rats Develop Autoimmune Diabetes Spontaneously and in Response to Environmental Perturbation