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《循环研究杂志》.2005年.第4期
 * Distinct NF-B Regulation by Shear Stress Through Ras-Dependent IB Oscillations
 * Essential Role of a Ca2+-Selective, Store-Operated Current (ISOC) in Endothelial Cell Permeability
 * Low-Voltage-Activated (T-Type) Calcium Channels Control Proliferation of Human Pulmonary Artery Myocytes
 * [Ca2+]i Reduction Increases Cellular Proliferation and Apoptosis in Vascular Smooth Muscle Cells
 * CCL2/Monocyte Chemoattractant Protein-1 Regulates Inflammatory Responses Critical to Healing Myocardial Infarcts
 * Contractile Properties of the Cultured Vascular Smooth Muscle Cells
 * Targeted Disruption of TGF-eCSmad3 Signaling Leads to Enhanced Neointimal Hyperplasia With Diminished Matrix Deposition in Response to Vascu
 * Homeobox Protein Hop Functions in the Adult Cardiac Conduction System
 * Immunomodulator FTY720 Induces eNOS-Dependent Arterial Vasodilatation via the Lysophospholipid Receptor S1P3
 * Lack of Myoglobin Causes a Switch in Cardiac Substrate Selection
 * Caveolin-1 Facilitates Mechanosensitive Protein Kinase B (Akt) Signaling In Vitro and In Vivo
 * Triadin Overexpression Stimulates Excitation-Contraction Coupling and Increases Predisposition to Cellular Arrhythmia in Cardiac Myocytes
 * Angiotensin II and Aldosterone Regulate Gene Transcription Via Functional Mineralocortocoid Receptors in Human Coronary Artery Smooth Muscle
 * NAD(P)H Oxidase Inhibition Attenuates Neuronal Chronotropic Actions of Angiotensin II
 * Endogenous p53 Protects Vascular Smooth Muscle Cells From Apoptosis and Reduces Atherosclerosis in ApoE Knockout Mice
 * Atherosclerotic Abdominal Aortic Aneurysm and the Interaction Between Autologous Human Plaque-Derived Vascular Smooth Muscle Cells, Type 1 N
 * Platelets and Chemokines in Atherosclerosis
 * A Role for Endoglin in Coupling eNOS Activity and Regulating Vascular Tone Revealed in Hereditary Hemorrhagic Telangiectasia
 * Common Variants in Myocardial Ion Channel Genes Modify the QT Interval in the General Population
 * Activation of Inflammation and Coagulation After Infusion of C-Reactive Protein in Humans
 * Regulation of Vascular Calcification
 * Role of Antioxidant-1 in Extracellular Superoxide Dismutase Function and Expression
 * Functional Effects of Rho-KinaseeCDependent Phosphorylation of Specific Sites on Cardiac Troponin
 * Impaired KCNQ1eCKCNE1 and Phosphatidylinositol-4,5-Bisphosphate Interaction Underlies the Long QT Syndrome
 * Increased Collagen Deposition and Diastolic Dysfunction but Preserved Myocardial Hypertrophy After Pressure Overload in Mice Lacking PKC
 * Enhancement of Cardiac Function and Suppression of Heart Failure Progression By Inhibition of Protein Phosphatase 1
 * Identification of Hypertension-Related Genes Through an Integrated Genomic-Transcriptomic Approach
 * Adenoviral Gene Transfer of FGF-5 to Hibernating Myocardium Improves Function and Stimulates Myocytes to Hypertrophy and Reenter the Cell Cy
 * Development of the Right Ventricular Inflow Tract and Moderator Band
 * SDF-1/CXCR4 Axis Is Instrumental in Neointimal Hyperplasia and Recruitment of Smooth Muscle Progenitor Cells
 * A Novel Form of Short QT Syndrome (SQT3) Is Caused by a Mutation in the KCNJ2 Gene
 * Spatial Heterogeneity of Endothelial Phenotypes Correlates With Side-Specific Vulnerability to Calcification in Normal Porcine Aortic Valves
 * Liver X Receptor Agonists Inhibit Cytokine-Induced Osteopontin Expression in Macrophages Through Interference With Activator Protein-1 Signa
 * Adenoviral Gene Transfer of Mutant Phospholamban Rescues Contractile Dysfunction in Failing Rabbit Myocytes With Relatively Preserved SERCA
 * NAD(P)H OxidaseeCDependent Self-Propagation of Hydrogen Peroxide and Vascular Disease
 * AMP-Activated Protein Kinase Signaling Stimulates VEGF Expression and Angiogenesis in Skeletal Muscle
 * Adenosine-Dependent Induction of Glutathione Peroxidase 1 in Human Primary Endothelial Cells and Protection Against Oxidative Stress
 * Hepatocyte Growth Factor Ameliorates the Progression of Experimental Autoimmune Myocarditis
 * Characterization of a Novel PKA Phosphorylation Site, Serine-2030, Reveals No PKA Hyperphosphorylation of the Cardiac Ryanodine Receptor in