现在位置：百拇书刊 > 国外期刊 > 循环研究杂志 > 2005年.第8期 《循环研究杂志》.2005年.第8期  * Transmural Heterogeneity of Na+eCCa2+ Exchange  * Prevention of Cardiac Hypertrophy by Atorvastatin in a Transgenic Rabbit Model of Human Hypertrophic Cardiomyopathy  * Optical Action Potential Upstroke Morphology Reveals Near-Surface Transmural Propagation Direction  * CD14+CD34low Cells With Stem Cell Phenotypic and Functional Features Are the Major Source of Circulating Endothelial Progenitors  * Interleukin-10 Suppresses Tissue Factor Expression in Lipopolysaccharide-Stimulated Macrophages via Inhibition of Egr-1 and a Serum Response  * Vascular Remodeling in Mice Lacking the Cytoplasmic Domain of Tissue Factor  * Protein Kinase G Transmits the Cardioprotective Signal From Cytosol to Mitochondria  * N488I Mutation of the 2-Subunit Results in Bidirectional Changes in AMP-Activated Protein Kinase Activity  * Distinct Pathways Regulate Expression of Cardiac Electrical and Mechanical Junction Proteins in Response to Stretch  * Thrombopoietin Gene TransfereCMediated Enhancement of Angiogenic Responses to Acute Ischemia  * Genome-Wide Screening for Target Regions of Histone Deacetylases in Cardiomyocytes  * Mitochondria-Derived Reactive Oxygen Species Dilate Cerebral Arteries by Activating Ca2+ Sparks  * Kv1.5 Surface Expression Is Modulated by Retrograde Trafficking of Newly Endocytosed Channels by the Dynein Motor  * Evidence in Favor of a Calcium-Sensing Receptor in Arterial Endothelial Cells  * Cardiomyocyte-Specific Knockout and Agonist of Peroxisome ProliferatoreCActivated Receptor- Both Induce Cardiac Hypertrophy in Mice  * Alk3/Bmpr1a Receptor Is Required for Development of the Atrioventricular Canal Into Valves and Annulus Fibrosus  * Combination of Tumor Necrosis Factor- Ablation and Matrix Metalloproteinase Inhibition Prevents Heart Failure After Pressure Overload in Tis  * Rapid Endothelial CelleCSelective Loading of Connexin 40 Antibody Blocks Endothelium-Derived Hyperpolarizing Factor Dilation in Rat Small Me  * Heterodimerization of 1- and 2-Adrenergic Receptor Subtypes Optimizes -Adrenergic Modulation of Cardiac Contractility  * Inhibition of Lipopolysaccharide-Induced Inflammatory Responses by an Apolipoprotein AI Mimetic Peptide  * Interdependent Serotonin Transporter and Receptor Pathways Regulate S100A4/Mts1, a Gene Associated With Pulmonary Vascular Disease  * Dual Serotonergic Regulation of Ventricular Contractile Force Through 5-HT2A and 5-HT4 Receptors Induced in the Acute Failing Heart  * Gene Transfer of Neuronal Nitric Oxide Synthase to Carotid Body Reverses Enhanced Chemoreceptor Function in Heart Failure Rabbits  * Phospholemman-Phosphorylation Mediates the -Adrenergic Effects on Na/K Pump Function in Cardiac Myocytes