《循环研究杂志》.2005年.第9期
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- PKC Activates eNOS and Increases Arterial Blood Flow In Vivo
- Effects of Estrogen Replacement Therapies on Mouse Platelet Function and Glycoprotein VI Levels
- Bone Morphogenetic Protein 4 Promotes Pulmonary Vascular Remodeling in Hypoxic Pulmonary Hypertension
- Sarco/Endoplasmic Reticulum Ca2+-ATPase Gene Transfer Reduces Vascular Smooth Muscle Cell Proliferation and Neointima Formation in the Rat
- Endothelial/Pericyte Interactions
- Asymmetric Dimethylarginine and the Risk of Cardiovascular Events and Death in Patients With Coronary Artery Disease
- Oral Small Peptides Render HDL Antiinflammatory in Mice and Monkeys and Reduce Atherosclerosis in ApoE Null Mice
- Expression and Phosphorylation of the Na-Pump Regulatory Subunit Phospholemman in Heart Failure
- Thyroid Hormone Targets Matrix Gla Protein Gene Associated With Vascular Smooth Muscle Calcification
- Transgenic Overexpression of Locally Acting Insulin-Like Growth Factor-1 Inhibits Ubiquitin-Mediated Muscle Atrophy in Chronic Left-Ventricu
- Impairment of Diazoxide-Induced Formation of Reactive Oxygen Species and Loss of Cardioprotection in Connexin 43 Deficient Mice
- Crosstalk of -Adrenergic Receptor Subtypes Through Gi Blunts -Adrenergic Stimulation of L-Type Ca2+ Channels in Canine Heart Failure
- Reduced Wall Compliance Suppresses Akt-Dependent Apoptosis Protection Stimulated by Pulse Perfusion
- Cell-Signaling Evidence for Adenosine Stimulation of Coronary Smooth Muscle Proliferation via the A1 Adenosine Receptor
- Transgenic Overexpression of Vascular Endothelial Growth Factor-B Isoforms by Endothelial Cells Potentiates Postnatal Vessel Growth In Vivo
- Increased Fibulin-5 and Elastin in S100A4/Mts1 Mice With Pulmonary Hypertension
- p47phox-Dependent NADPH Oxidase Regulates Flow-Induced Vascular Remodeling
- Targeting of a Novel Ca+2/Calmodulin-Dependent Protein Kinase II Is Essential for Extracellular Signal-Regulated KinaseeCMediated Signaling
- The Role of the Multidrug Resistance Protein-1 in Modulation of Endothelial Cell Oxidative Stress
- A Myocardial Nox2 Containing NAD(P)H Oxidase Contributes to Oxidative Stress in Human Atrial Fibrillation
- Explaining the Phenomenon of Nitrate Tolerance
- Enhanced Functional Gap Junction Neoformation by Protein Kinase AeCDependent and Epac-Dependent Signals Downstream of cAMP in Cardiac Myocyt
- Glycogen Synthase Kinase 3 Inhibits Myocardin-Dependent Transcription and Hypertrophy Induction Through Site-Specific Phosphorylation
- Protein Kinase CeCDependent Phosphorylation of Syndecan-4 Regulates Cell Migration
- Cardiac Stem Cells Possess Growth Factor-Receptor Systems That After Activation Regenerate the Infarcted Myocardium, Improving Ventricular F
- Loss of Pentameric Symmetry in C-Reactive Protein Induces Interleukin-8 Secretion Through Peroxynitrite Signaling in Human Neutrophils
- Liver X Receptor Activation Controls Intracellular Cholesterol Trafficking and Esterification in Human Macrophages
- Intermittent Hypoxia Induces Hyperlipidemia in Lean Mice
- Arrestin-Independent Internalization and Recycling of the Urotensin Receptor Contribute to Long-Lasting Urotensin IIeCMediated Vasoconstrict
- Vascular Neointimal Formation and Signaling Pathway Activation in Response to Stent Injury in Insulin-Resistant and Diabetic Animals
- Complement Activation in Angiotensin IIeCInduced Organ Damage
- Alterations in Internal Elastic Lamina Permeability As a Function of Age and Anatomical Site Precede Lesion Development in Apolipoprotein Ee
- Endothelial Protein Kinase C Isoform Identity and Differential Activity of PKC in an Athero-Susceptible Region of Porcine Aorta
- Aldosterone and Angiotensin II Synergistically Induce Mitogenic Response in Vascular Smooth Muscle Cells
- Cardiac-Specific Loss of N-Cadherin Leads to Alteration in Connexins With Conduction Slowing and Arrhythmogenesis
- Paradoxical Cellular Ca2+ Signaling in Severe but Compensated Canine Left Ventricular Hypertrophy