现在位置：百拇书刊 > 国外期刊 > 循环研究杂志 > 2005年.第11期 《循环研究杂志》.2005年.第11期  * Progesterone Antagonizes the Vasoprotective Effect of Estrogen on Antioxidant Enzyme Expression and Function  * Dual Pathways for Nuclear Factor B Activation by Angiotensin II in Vascular Smooth Muscle  * Embryonic even skippedeCDependent Muscle and Heart Cell Fates Are Required for Normal Adult Activity, Heart Function, and Lifespan  * Haplotype Effect of the Matrix Metalloproteinase-1 Gene on Risk of Myocardial Infarction  * Discrepancies Between Nitroglycerin and NO-Releasing Drugs on Mitochondrial Oxygen Consumption, Vasoactivity, and the Release of NO  * NADPH Oxidase Activity and Function Are Profoundly Greater in Cerebral Versus Systemic Arteries  * Synthetic Retinoid Am80 Suppresses Smooth Muscle Phenotypic Modulation and In-Stent Neointima Formation by Inhibiting KLF5  * Nitric Oxide Regulates Transforming Growth Factor- Signaling in Endothelial Cells  * Nuclear RNA Foci in the Heart in Myotonic Dystrophy  * Impaired CXCR4 Signaling Contributes to the Reduced Neovascularization Capacity of Endothelial Progenitor Cells From Patients With Coronary  * Cardiac Myosin-Binding Protein-C Phosphorylation and Cardiac Function  * Myocardin Enhances Smad3-Mediated Transforming Growth Factor-1 Signaling in a CArG Box-Independent Manner  * Activation of the Endothelial Store-Operated ISOC Ca2+ Channel Requires Interaction of Protein 4.1 With TRPC4  * Effects of D-4F on Vasodilation and Vessel Wall Thickness in Hypercholesterolemic LDL ReceptoreCNull and LDL Receptor/Apolipoprotein A-I Dou  * Ischemic Neoangiogenesis Enhanced by 2-Adrenergic Receptor Overexpression  * Enhanced Store OverloadeCInduced Ca2+ Release and Channel Sensitivity to Luminal Ca2+ Activation Are Common Defects of RyR2 Mutations Linked  * Bone Morphogenetic Protein Signaling Modulates Myocardin Transactivation of Cardiac Genes  * FcRIIB Mediates C-Reactive Protein Inhibition of Endothelial NO Synthase  * Ca2+ InfluxeCInduced Sarcoplasmic Reticulum Ca2+ Overload Causes Mitochondrial-Dependent Apoptosis in Ventricular Myocytes  * Calreticulin Destabilizes Glucose Transporter-1 mRNA in Vascular Endothelial and Smooth Muscle Cells Under High-Glucose Conditions  * Platelet-Derived Growth Factor D Induces Cardiac Fibrosis and Proliferation of Vascular Smooth Muscle Cells in Heart-Specific Transgenic Mic  * Mobilization of Bone MarroweCDerived Cells Enhances the Angiogenic Response to Hypoxia Without Transdifferentiation Into Endothelial Cells  * Intrasarcoplasmic Amyloidosis Impairs Proteolytic Function of Proteasomes in Cardiomyocytes by Compromising Substrate Uptake