现在位置：百拇书刊 > 国外期刊 > 循环研究杂志 > 2005年.第12期 《循环研究杂志》.2005年.第12期  * Gab1, SHP2, and Protein Kinase A Are Crucial for the Activation of the Endothelial NO Synthase by Fluid Shear Stress  * Ca2+/CalmodulineCDependent Protein Kinase Modulates Cardiac Ryanodine Receptor Phosphorylation and Sarcoplasmic Reticulum Ca2+ Leak in Heart  * Increased Neointima Formation in Cysteine-Rich Protein 2eCDeficient Mice in Response to Vascular Injury  * Targeted Deletion of Kv4.2 Eliminates Ito,f and Results in Electrical and Molecular Remodeling, With No Evidence of Ventricular Hypertrophy  * Nuclear Targeting of Akt Enhances Ventricular Function and Myocyte Contractility  * PTEN Activity Is Modulated During Ischemia and Reperfusion  * Soluble Vascular Endothelial Growth Factor Receptor-1 (sFLT-1) Mediates Downregulation of FLT-1 and Prevents Activated Neutrophils From Wome  * Adiponectin Inhibits Endothelial Synthesis of Interleukin-8  * Protective Effect of the KCNMB1 E65K Genetic Polymorphism Against Diastolic Hypertension in Aging Women and Its Relevance to Cardiovascular  * TRPV4 Forms a Novel Ca2+ Signaling Complex With Ryanodine Receptors and BKCa Channels  * Oxidative Mediated Lipid Peroxidation Recapitulates Proarrhythmic Effects on Cardiac Sodium Channels  * cAMP-Binding Protein Epac Induces Cardiomyocyte Hypertrophy  * ExcitationeCContraction Coupling in Na+eCCa2+ Exchanger Knockout Mice  * Contribution of Kv Channels to Phenotypic Remodeling of Human Uterine Artery Smooth Muscle Cells  * Phospholipase C Modulates -Adrenergic ReceptoreC Dependent Cardiac Contraction and Inhibits Cardiac Hypertrophy