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冠状动脉微循环障碍加速大鼠心肌梗死后不良心室重构的机制研究(4)
http://www.100md.com 2017年12月1日 《新医学》 201712
     [6]Pereira FE, Cronin C, Ghosh M, Zhou SY, Agosto M, Subramani J, Wang R, Shen JB, Schacke W, Liang B, Yang TH, McAulliffe B, Liang BT, Shapiro LH.CD13 is essential for inflammatory trafficking and infarct healing following permanent coronary artery occlusion in mice.Cardiovasc Res,2013,100(1):74-83.

    [7]Chen W, Frangogiannis NG.Fibroblasts in post-infarction inflammation and cardiac repair.Biochim Biophys Acta,2013,1833(4):945-953.

    [8]Frangogiannis NG.The inflammatory response in myocardial injury, repair, and remodelling.Nat Rev Cardiol,2014,11(5):255-265.

    [9]肖月瓊,周万兴. 金属蛋白酶组织抑制因子-1对心室重构影响的研究进展. 新医学,2012, 43(7):515-518.

    [10]Zhang J, Li XX, Bian HJ, Liu XB, Ji XP, Zhang Y.Inhibition of the activity of Rho-kinase reduces cardiomyocyte apoptosis in heartischemia/reperfusion via suppressing JNK-mediated AIF translocation.Clin Chim Acta,2009,401(1-2):76-80.

    [11]Frangogiannis NG.The immune system and the remodeling infarcted heart: cell biological insights and therapeuticopportunities.J Cardiovasc Pharmacol,2014,63(3):185-195.

    [12]Seropian IM, Toldo S, Van Tassell BW, Abbate A.Anti-inflammatory strategies for ventricular remodeling following ST-segment elevation acutemyocardial infarction.J Am Coll Cardiol,2014,63(16):1593-1603.

    [13]Frangogiannis NG.Regulation of the inflammatory response in cardiac repair.Circ Res,2012,110(1):159-173.

    (收稿日期:2017-07-28)

    (本文编辑:林燕薇), 百拇医药(郭文玉 彭长农 柯晓 邹春霞 王小庆)
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