当前位置: 首页 > 期刊 > 《中国美容医学》 > 2012年第15期 > 正文
编号:12334020
白癜风发病机制及治疗策略(2)
http://www.100md.com 2012年8月1日 高天文 刘玲
第1页

    参见附件。

     [9]Quan C,Ren YQ,Xiang LH,et al.Genome-wide association study for vitiligo identifies susceptibility loci at 6q27 and the MHC[J].Nat Genet,2010,42(7):614-618.

    [10]高天文.挑战白癜风治疗(述评)[J].第四军医大学学报,2001,22:2213-2215.

    [11]Le Poole IC, Das PK, van den Wijngaard RM,et al.Review of the etiopathomechanism of vitiligo: a convergence theory[J]. Exp Dermatol, 1993,2(4):145-153.

    [12]van den Boorn JG,Konijnenberg D,Dellemijn TA,et al.Autoimmune destruction of skin melanocytes by perilesional T cells from vitiligo patients[J].J Invest Dermatol,2009, 129:2220-2232.

    [13]Garbelli S,Mantovani S,Palermo B,et al.Melanocyte-specific, cytotoxic T cell responses in vitiligo:the effective variant of melanoma immunity[J]? Pigment Cell Res,2005,18:234-242.

    [14]Schallreuter KU,Wood JM,Berger J.Low catalase levels in the epidermis of patients with vitiligo[J].J Invest Dermatol,1991,97:1081-1085.

    [15]Pelle E, Mammone T, Maes D,et al.Keratinocytes act as a source of reactive oxygen species by transferring hydrogen peroxide to melanocytes[J].J Invest Dermatol,2005,124:793-797.

    [16]Zhou Z,Li CY,Li K,et al.Decreased methionine sulphoxide reductase A expression renders melanocytes more sensitive to oxidative stress: a possible cause for melanocyte loss in vitiligo[J].Br J Dermatol,2009 161(5):504-509.

    [17]Schallreuter KU,Rubsam K,Gibbons NC,et al.Methionine sulfoxidereductases A and B are deactivated by hydrogen peroxide (H2O2) in the epidermis of patients with vitiligo[J].J Invest Dermatol,2008,128:808-815.

    [18]Sravani PV,Babu NK,Gopal KV,et al.Determination of oxidative stress in vitiligo by measuring superoxide dismutase and catalase levels in vitiliginous and non-vitiliginous skin[J] ......

您现在查看是摘要介绍页,详见PDF附件(1680kb)