骨折患者血浆TNF-α、IL-10 动态变化及其意义(2)
第1页 |
参见附件(264KB,2页)。
参考文献:
[1]王正国.创伤医学基础[M]. 长春:吉林科学技术出版社,1999.12.
[2]吴孟超,仲剑平.外科学新理论新技术[M].上海:上海学技术教育出版社,1996.17.
[3]姚永明,盛志勇.脓毒症信号转导机制的现代认识[J].中国危重病急救医学,2003,15(1):3.
[4]Hagimoto N,Kuwomo K,Nomoto Y,et al. Apoptosis and expression of Fas/Fas ligand mRNA in bleomycin- induced pulmonary fibrosis in mice[J]. Am JRespir Cell Mol Biol,1997,16:91.
[5]Grassme H,Kirschnck S,Rieth mueller J,et al. CD95/CD95 ligand inter actionsonepithelialcells in host defense to psuedomon as aeruginosa[J].Science,2003,29:227.
[6]Spielmann S,Kerner T,Ahlers O,et al. Early detection of in-creased tumour necrosis factor alpha(TNF-α) and soluble TNF receptor pro-tein plasma levels after trauma reveals association with the clinical coursed [J].Acta Anaesth Scand,2001,45(3):364.
[7]PapeHC,Van Griensvern M,Rice J,et al. Major secondary surgery in blunt trauma patients and perioperative cytokine liberation:deter-mination of the clinical relevance of biochemical markers[J].J Trauma,2001,50(6):989.
[8]Jedynak M,Siemiatkowski A.The role of monocytes macrophages and their cytokines in the development of munosuppression after severe injury[J]. Pol Merkuriusz Lek,2002,13(75):238.
收稿日期:2007-03-07
注:本文中所涉及到的图表、注解、公式等内容请以PDF格式阅读原文。
[ 上 页 ]
您现在查看是摘要介绍页,详见PDF附件(264KB,2页)。