枸橼酸铁铵对小胶质细胞激活状态的影响(4)
[26] ZHAN Y, PAOLICELLI R, SFORAZZINI F, et al. Deficient neuron-microglia signaling results in impaired functional brain connectivity and social behavior[J]. Nature Neuroscience, 2014,17(3):400-406.
[27] SCHAFER D P, LEHRMAN E K, KAUTZMAN A G, et al. Microglia sculpt postnatal neural circuits in an activity and complement-dependent manner[J]. Neuron, 2012,74(4):691-705.
[28] PAOLICELLI R C, BOLASCO G, PAGANI F, et al. Synaptic pruning by microglia is necessary for normal brain development[J]. Science, 2011,333(648):1456-1458.
[29] JOERS V, TANSEY M G, MULAS G, et al. Microglial phenotypes in Parkinson’s disease and animal models of the di-sease[J]. Prog Neurobiol, 2017,155:57-75.
[30] HANISCH U K, KETTENMANN H. Microglia: active sensor and versatile effector cells in the normal and pathologic brain[J]. Nature Neuroscience, 2007,10(11):1387-1394.
[31] SIERRA A, ABIEGA O, SHAHRAZ A, et al. Janus-faced microglia: beneficial and detrimental consequences of microg-lial phagocytosis[J]. Front Cell Neurosci, 2013,7:6.
(本文編辑 马伟平), http://www.100md.com(于小淇 谢俊霞 徐华敏)
[27] SCHAFER D P, LEHRMAN E K, KAUTZMAN A G, et al. Microglia sculpt postnatal neural circuits in an activity and complement-dependent manner[J]. Neuron, 2012,74(4):691-705.
[28] PAOLICELLI R C, BOLASCO G, PAGANI F, et al. Synaptic pruning by microglia is necessary for normal brain development[J]. Science, 2011,333(648):1456-1458.
[29] JOERS V, TANSEY M G, MULAS G, et al. Microglial phenotypes in Parkinson’s disease and animal models of the di-sease[J]. Prog Neurobiol, 2017,155:57-75.
[30] HANISCH U K, KETTENMANN H. Microglia: active sensor and versatile effector cells in the normal and pathologic brain[J]. Nature Neuroscience, 2007,10(11):1387-1394.
[31] SIERRA A, ABIEGA O, SHAHRAZ A, et al. Janus-faced microglia: beneficial and detrimental consequences of microg-lial phagocytosis[J]. Front Cell Neurosci, 2013,7:6.
(本文編辑 马伟平), http://www.100md.com(于小淇 谢俊霞 徐华敏)