当前位置: 首页 > 期刊 > 《医药产业资讯》 > 2007年第15期
编号:11479806
线粒体ATP敏感性钾通道与心肌保护(4)
http://www.100md.com 2007年8月17日 林永文 谭建新
第1页

    参见附件(460KB,3页)。

     [25]Xia Y, Rajapurohitam V, Cook MA, Karmazyn M. Inhibition of phenylephrine induced hypertrophy in rat neonatal cardiomyocytes by the mitochondrial KATP channel opener diazoxide[J].J Mol Cell Cardiol, 2005,38(2):237-239.

    [26]Ichinose M,Yonemochi H,Sato T, et al. Diazoxide triggerscardioprotectin against apopto sis induced by oxidativestress[J].Am J Physiol Heart Circ Physiol,2003,284(6):H2235-H2241.

    [27]Xu MF,Wang YG,Ayub A, et al.Mitochondrial KATP channel activation reduces anoxic injury by restoring mitochondrial membrane potential[J]. Am J Physiol Heart Circ Physiol,2001,281(3):H1295-H1303.

    [28]Akao M, Ohler A, O'Rourke B,et al.Mitochondrial ATP-sensitive potassium channels inhibit apoptosis induced by oxidative stress in cardiac cells [J]. Circ Res, 2001, 88 (12): 1267-1275.

    [29]McCully JD,Wakiyama H, Cowan DB,et al.Diazoxide amelioration of myocardial injury and mitochondrial damage during cardiac surgery [J]. Ann Thorac Surg, 2002, 74 (6): 2138-2145.

    (收稿日期:2007-02-25)

    本文中所涉及到的图表、注解、公式等内容请以PDF格式阅读原文。

    [ 上 页 ] [ 下 页 ]

您现在查看是摘要介绍页,详见PDF附件(460KB,3页)