原发性高血压患者血压变异性与血清TNF-α、IL-6、hs-CRP关系的研究(3)
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参见附件。
[6] Eto M，Toba K，Akishita M，et al. Reduced endothelial vasomotor function and enhanced neointimal formation after vascular injury in a rat model of blood pressure lability [J]. Hypertens Res，2003，26(12)：991-998.
[7] Abramson JL，Lewis C，Murrah NV，et al. Relation of C-reactive protein and tumor necrosis factor-alpha to ambulatory blood pressure variability in healthy adults [J]. Am J Cardiol，2006，98(5)：649-652.
[8] Bautista LE，Vera LM，Arenas IA，et al. Independent association between inflammatory markers (C-reactive protein， interleukin-6， and TNF-alpha) and essential hypertension [J]. J Hum Hypertens，2005，19(2)：149-154.
[9] Gabay C，Kushner I. Acute-phase proteins and other systemic responses to inflammation [J]. N Engl J Med，1999，340(17)：448-454.
[10] Sesso HD，Buring JE，Rifai N，et al. C-reactive protein and the risk of developing hypertension [J]. JAMA，2003，290(22)：3000-3002.
[11] Blake GJ，Rifai N，Buring JE，et al. Blood pressure，C-reactive protein， and risk of future cardiovascular events [J]. Circulation，2007，115(20)： 537-539.
[12] Festa A ，Agostino RD，Tracy RP，et al. Elevated levels of acute- phase protein and plasminogen activator inhibitor-1 predict the development of type 2 diabetes [J]. Diabetes，2002，51(4)：1131-1137.
(收稿日期：2012-01-05 本文编辑：程 铭)
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