丝裂原活化蛋白激酶与缺血性脑血管病的关系(3)
[5] Sun J，Nan G.The Mitogen-Activated Protein Kinase (MAPK) Signaling Pathway as a Discovery Target in Stroke[J].Journal of Molecular Neuroscience，2016，59(1)：90-98.
[6] Chan L P，Liu C，Chiang F Y，et al.IL-8 promotes inflammatory mediators and stimulates activation of p38 MAPK/ERK-NF-κB pathway and reduction of JNK in HNSCC[J].Oncotarget，2017，8(34).
[7] Jiang S Y，Zou Y Y，Wang J T.p38 mitogen-activated protein kinase–induced nuclear factor kappa-light-chain-enhancer of activated B cell activity is required for neuroprotection in retinal ischemia/reperfusion injury[J].Molecular Vision，2012，18(220-25)：2096.
, http://www.100md.com
[8] Bogoyevitch M A，Kobe B.Uses for JNK：the many and varied substrates of the c-Jun N-terminal kinases[J].Microbiology & Molecular Biology Reviews Mmbr，2006，70(4)：1061.
[9] Wang Y，Hai B，Niu X，et al.Chronic intermittent hypoxia disturbs insulin secretion and causes pancreatic injury via MAPK signaling pathway[J].Biochem Cell Biol，2017，95(3)：415-420.
[10] Courtial L，Picco V，Grover R，et al.The c-Jun N-terminal kinase prevents oxidative stress induced by UV and thermal stresses in corals and human cells[J].Scientific Reports，2017，7：45713.
, http://www.100md.com
[11] Han M S，Barrett T，Brehm M A，et al.Inflammation Mediated by JNK in Myeloid Cells Promotes the Development of Hepatitis and Hepatocellular Carcinoma[J].Cell Reports，2016，15(1)：19.
[12] Lai T W，Zhang S，Wang Y T.Excitotoxicity and stroke：identifying novel targets for neuroprotection[J].Progress in Neurobiology，2014，115(2)：157.
[13] Xiao L，Feng C，Chen Y.Glucocorticoid rapidly enhances NMDA-evoked neurotoxicity by attenuating the NR2A-containing NMDA receptor-mediated ERK1/2 activation[J].Molecular Endocrinology，2010，24(3)：497-510.
, http://www.100md.com
[14] Poddar R，Paul S.Novel crosstalk between ERK MAPK and p38 MAPK leads to homocysteine-NMDA receptor-mediated neuronal cell death[J].Journal of Neurochemistry，2013，124(4)：558-570.
[15] Navon H，Bromberg Y，Sperling O，et al.Neuroprotection by NMDA preconditioning against glutamate cytotoxicity is mediated through activation of ERK 1/2， inactivation of JNK， and by prevention of glutamate-induced CREB inactivation[J].Journal of Molecular Neuroscience，2012，46(1)：100-108.
[16] Lee K M，Lean A G M.New advances on glial activation in health and disease[J].World Journal of Virology，2015，4(2)：42-55.
[17] Park J，Min J S，Kim B，et al.Mitochondrial ROS govern the LPS-induced pro-inflammatory response in microglia cells by regulating MAPK and NF-κB pathways[J].Neuroscience Letters，2015，584：191-196., 百拇医药(王锐 胡风云 赵晓霞 蔚洪恩)
[6] Chan L P，Liu C，Chiang F Y，et al.IL-8 promotes inflammatory mediators and stimulates activation of p38 MAPK/ERK-NF-κB pathway and reduction of JNK in HNSCC[J].Oncotarget，2017，8(34).
[7] Jiang S Y，Zou Y Y，Wang J T.p38 mitogen-activated protein kinase–induced nuclear factor kappa-light-chain-enhancer of activated B cell activity is required for neuroprotection in retinal ischemia/reperfusion injury[J].Molecular Vision，2012，18(220-25)：2096.
, http://www.100md.com
[8] Bogoyevitch M A，Kobe B.Uses for JNK：the many and varied substrates of the c-Jun N-terminal kinases[J].Microbiology & Molecular Biology Reviews Mmbr，2006，70(4)：1061.
[9] Wang Y，Hai B，Niu X，et al.Chronic intermittent hypoxia disturbs insulin secretion and causes pancreatic injury via MAPK signaling pathway[J].Biochem Cell Biol，2017，95(3)：415-420.
[10] Courtial L，Picco V，Grover R，et al.The c-Jun N-terminal kinase prevents oxidative stress induced by UV and thermal stresses in corals and human cells[J].Scientific Reports，2017，7：45713.
, http://www.100md.com
[11] Han M S，Barrett T，Brehm M A，et al.Inflammation Mediated by JNK in Myeloid Cells Promotes the Development of Hepatitis and Hepatocellular Carcinoma[J].Cell Reports，2016，15(1)：19.
[12] Lai T W，Zhang S，Wang Y T.Excitotoxicity and stroke：identifying novel targets for neuroprotection[J].Progress in Neurobiology，2014，115(2)：157.
[13] Xiao L，Feng C，Chen Y.Glucocorticoid rapidly enhances NMDA-evoked neurotoxicity by attenuating the NR2A-containing NMDA receptor-mediated ERK1/2 activation[J].Molecular Endocrinology，2010，24(3)：497-510.
, http://www.100md.com
[14] Poddar R，Paul S.Novel crosstalk between ERK MAPK and p38 MAPK leads to homocysteine-NMDA receptor-mediated neuronal cell death[J].Journal of Neurochemistry，2013，124(4)：558-570.
[15] Navon H，Bromberg Y，Sperling O，et al.Neuroprotection by NMDA preconditioning against glutamate cytotoxicity is mediated through activation of ERK 1/2， inactivation of JNK， and by prevention of glutamate-induced CREB inactivation[J].Journal of Molecular Neuroscience，2012，46(1)：100-108.
[16] Lee K M，Lean A G M.New advances on glial activation in health and disease[J].World Journal of Virology，2015，4(2)：42-55.
[17] Park J，Min J S，Kim B，et al.Mitochondrial ROS govern the LPS-induced pro-inflammatory response in microglia cells by regulating MAPK and NF-κB pathways[J].Neuroscience Letters，2015，584：191-196., 百拇医药(王锐 胡风云 赵晓霞 蔚洪恩)
参见：首页 > 医疗版 > 疾病专题 > 神经内科 > 脑血管病