【摘要】酒精性肝病(alcoholic liver disease，ALD)是长期过量饮酒所导致的肝损害。已知乙醇进入肝细胞后，经过肝乙醇脱氢酶、过氧化氢体分解酶和肝细胞微粒体乙醇氧化酶系2条途径氧化为乙醛。过量饮酒，大量乙醛对肝细胞有明显的毒性作用，直接或间接导致肝细胞变性、坏死以及纤维化，严重时可发展为肝硬化、肝癌。酒精性肝硬化为酒精性肝病进一步发展所致，对丹参酮ⅡA治愈两例酒精性肝硬化合并腹水的临床治疗进行分析。

    【关键词】酒精性肝硬化；丹参酮ⅡA

    【Abstract】Alcoholic liver disease (alcoholic liver disease, ALD)is caused by long-term excessive alcohol liver damage.Ethanol into the liver cells are known,after the liver alcoholdehydrogenase, hydrogen peroxide decomposition activity and liver microsomal ethanol oxidation enzymes in two ways oxidized to acetaldehyde. Excessive drinking, a large number of liver cells acetaldehyde has obvious toxicity, either directly or indirectly lead to liver cell degeneration, necrosis and fibrosis, severe cases can develop into cirrhosis, liver cancer. Alcoholic cirrhosis is (Alcoholic cirrhosis)due to the further development of alcoholic liver disease, this paper two cases of alcoholic cirrhosis with ascites clinical analysis as follows.

    【Key words】alcoholic cirrhosis tanshinoneⅡA

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